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Find out how to reduce your chances of developing cancer by 80%. Dr Coleman lists the foods which can cause - and prevent - cancer.

POWER OVER CANCER

Vernon Coleman

CONTENTS

Foreword

Preface

Part One
The Cancer Industry: Misdirected and Ineffective

• Chapter 1: The War Against Cancer

• Chapter 2: Women Golfers and Breast Cancer

• Chapter 3: Animal Experiments: More Harm Than Good

• Chapter 4: The Search for a Magic Cure

• Chapter 5: Losing the War

• 1: The Top Ten Known Causes of Cancer

• 2: The Early Warning Signs of Cancer

• 3: Fighting Cancer

• 4: Breast Cancer

NOTE

This book is not intended as an alternative to personal, professional medical advice. The reader should consult a physician in all matters relating to health, and particularly in respect of any symptoms which may require diagnosis or medical attention.

While the advice and information in this book are believed to be accurate at the time of going to press, neither the author nor the publisher can accept any legal responsibility or liability for any errors or omissions that may be made.

FOREWORD

I started to research this subject for my own benefit. Since qualifying as a doctor just over two decades ago I have read much conflicting information about cancer and I wanted to find out the truth for myself. I knew that I should follow a diet that contained natural fibre and that was low in fat but I wanted to know more. I wanted to make sure that I ate a diet that would give me the best possible chance of avoiding cancer. I wanted to find out what researchers had discovered in the last two decades.

The more I studied, and the more I searched for information, the more convinced I became that the truth about the links between food and cancer had been deliberately suppressed by industries determined to continue to sell their existing products (whatever the human cost might be), by politicians committed to supporting those industries and by a medical profession which has become a pawn of the pharmaceutical industry.

You may be as surprised as I was to find that the knowledge that there is a link between food and cancer is not new. Way back in January 1892 Scientific American magazine reported: 'cancer is most frequent among those branches of the human race where carnivorous habits prevail'. Even before that, in 1804, J Abernethy, in Surgical Observations on Tumours reported on the effectiveness of a vegetarian diet as a treatment for cancer. Indeed, according to a report published in 1988 by the United States Department of Health and Human Services, called The Surgeon General's Report on Nutrition and Health, diet has been suspected as a cause of cancer since the disease was recognised in the first century A.D. Empirical evidence was first reported in 1913 and studies were published over half a century ago demonstrating the links between food and cancer. In 1933 researchers suggested that wholemeal bread and cruciferous vegetables helped to protect against cancer and in 1940 insurance company records suggested that overweight individuals were more likely to develop cancer than normal or underweight people. This research led to plenty of experiments involving animals (lots of researchers altered the dietary habits of animals to find out whether or not they were more or less likely to develop cancer) but no one seemed too concerned to apply the knowledge that had been obtained from a study of human beings to human beings, and little effort was made to conduct further epidemiological studies.

In Nutrition and Health, the report published in 1988 by the United States Surgeon General, a table was published suggesting that diet could be responsible for up to 70% of all cancers - with a (conservative) 'best estimate' suggesting that diet was responsible for 35% of all cancers.

The Surgeon General suggested five possible mechanisms to explain the 'observed association between diet, digestive processes and cancer'. These were:

1. The presence of carcinogens in food. These could be present naturally, could be contaminants or could be formed as a result of cooking or preserving the food.

2. Carcinogens within the body might be activated or deactivated by something in the diet. For example, the beta carotene present in some foods may slow down or block potentially cancer inducing processes.

3. Carcinogens may be formed within the body. For example, naturally produced bile acids, excreted to help deal with consumed fats, may be converted into tumour producing chemicals by bacteria which live in the colon. The bacteria may be affected by diet.

4. The cancer process may be enhanced by some foods (for example fats) or may be inhibited by some foods (for example, vitamin A).

5. An imbalance of nutrients may impair the immunity of the body and may, therefore, affect the ability of the body to defend itself - either by repairing damaged DNA or by rejecting malignant cells.

I had not intended to write this book when I started my researches. But having discovered what I have discovered I had no choice but to publish a book containing my findings. Although the bulk of this book deals with food (partly because food is the main cause of cancer and partly because there is more confusion and misinformation about the links between food and cancer than there is, say, about the links between tobacco and cancer) I have also included information about most of the other well known or suspected links. I do not, however, suggest that the list of possible causes of cancer in this book is comprehensive. I have dealt with the causes which attract most attention and which I regard both as being of most significance and easiest to do something about.

Cancer inducing chemicals are all around us - in air polluted with tobacco smoke and chemicals, in food and in the chemical products with which our homes are crammed. Our bodies are constantly producing potentially cancerous cells. We can help to protect ourselves against cancer by avoiding the obvious carcinogens (such as tobacco, fat and meat) but we can also build up our defence systems, and protect ourselves against cancer, by eating the right foods.

Finally, I think it is appropriate to point out that my own eating habits certainly follow the advice in this book. In other words 'I practise what I preach'! I believe that the earlier you adopt the eating habits recommended in this book the lower your cancer risk will be. But I also believe that it is never too late to change.

Vernon Coleman 1995

PREFACE

Cancer is not a single disease. It is a word which describes a great many quite different diseases. The one thing they all have in common is that there is an uncontrolled and disorderly growth of abnormal cells. It is quite normal for cells to grow and to reproduce. Every minute, in every human body, an astonishing ten million cells divide. Usually everything goes well. The cells divide in the right way and at the right time. But when a cell becomes a 'cancer cell' it grows and divides at an abnormally rapid rate. These abnormal 'cancer cells' destroy or push aside the normal, functioning cells. If the 'cancer cells' are not stopped they may spread to other parts of the body and take up residence in other, different organs. 'Cancer cells' may be carried around the body through the blood vessels or the lymph channels. When a cancer spreads and appears in another part of the body the new growths are known as metastases. Cancer can also spread by 'crab-like' outgrowths (hence the name 'cancer'). It is now widely accepted that many of the cancers which most commonly occur develop because outside substances trigger some sort of reaction - and cancer development - in the body. This book is much concerned with those 'triggers'.

Cancer is not the unknown, dark shrouded mystery killer which it is often thought to be. We do not know enough to recommend a diet which will enable all individuals to avoid all food related cancers. But we know enough to make a difference. If we make the decision to avoid those foods and other substances which research has shown can lead to the development of cancer, and to eat those foods which can strengthen our defences against cancer, then I believe that we can dramatically influence our susceptibility to the disease.

It is fairly wide known, I think, that cigarettes, sunshine, asbestos and X rays cause cancer. But it is less widely known that fatty foods and meat cause cancer. And it is less well known that fibre and green vegetables help protect against cancer.

People get cancer through bad luck and inherited susceptibilities. But they also get it through ignorance - ignorance which has been deliberately sustained through commercial greed and political expediency. When businessmen, politicians and doctors know how to prevent 80% of cancers but do nothing it is difficult to avoid the conclusion that those cancer deaths are deliberate. Eight out of ten people who die from cancer have effectively been murdered in cold blood. It is difficult to avoid the conclusion that cancer is now often a disease of choice - but the choice is made for us by those whose sense of responsibility does not match their authority. For too many people the choice is made by politicians, businessmen and doctors who do not understand that with knowledge comes responsibility.

In the Proceedings of the Nutrition Society in 1990, Sir Richard Doll said: 'There is now uniform agreement among oncologists that the incidence of cancer is determined, in large part, by factors in the environment and aspects of behaviour that are capable of modification or avoidance.'

For decades much of the international 'cancer industry' has been devoted to finding magical 'cures' for cancer - with little useful effect. But since many of the causes of cancer are known my aim in this book is quite simply to explain those causes and to tell you how you can best cut your own risk of developing cancer substantially.

Changing large organisations can take decades and generations. I have absolutely no doubt that in a few decades time eating meat, drinking milk and smoking tobacco will be regarded as bizarre and reckless practices. Our descendants will not understand why we exposed ourselves to such unnecessary risks. But it will take time for those changes to take place. There are laboratories to dismantle and companies to close down; butchers' shops to shut and farmers to direct away from animal farming and towards the growing of healthy crops. With so many vested interests to oppose all this will take a long time.

But you do not have to wait for the authorities to tell you what to eat and what to avoid eating. You do not have to wait for the food companies to start selling foods that will limit your chances of developing cancer. You don't have to wait for the cancer industry to turn its attention away from pointless and misleading animal experiments and start telling you what to eat to reduce your cancer risk. You can take action now. This book will tell you how you can help yourself reduce your cancer risk by up to 80%. Since we know what causes around 80% of cancers it is clear that we also know how to avoid 80% of cancers. I cannot and do not guarantee that you won't get cancer if you follow the advice in this book. But I do firmly believe that your chances of developing cancer will be significantly lower than it will be if you take no action to protect yourself. And I also believe that if you do develop cancer then the advice in this book will improve your chances of fighting your cancer and winning.

Vernon Coleman, 1995

CHAPTER ONE

THE WAR AGAINST CANCER

Cancer is, along with heart disease and stroke, one of the three big killers of our time. But cancer is the killer that frightens people most. The very word is so emotive that most doctors try not to use it when talking to patients. Instead of talking of cancer they talk of 'tumours' and 'growths'. They know that cancer is something most people don't even like to talk about.

Cancer of the colon is a much bigger killer than AIDS but you won't see many show business fund raisers being organised to help colon cancer sufferers in the way that money is raised to help AIDS sufferers (although money is raised - in vast quantities - to help pay for the salaries and expenses of researchers looking for a cure for cancer).

Until it touches them or someone in their family most people try not to think about cancer. When they see a collecting tin for a cancer charity they may pop in a few coins in the belief that by making a donation to a cancer charity they are helping to conquer the disease. Putting money into a cancer charity collecting tin is like throwing coins into a wishing well; it is the twentieth century equivalent of a good luck charm.

In order to ensure that money continues to pour in the cancer industry must persuade potential contributors and supporters that it is making progress in the fight against cancer. Like all large organisations the cancer industry needs a vast quantity of money just to remain in business; there are salaries to be paid, rents and rates to be paid, electricity bills to be paid, advertising and public relations departments to pay for and so on.

To help in this aim the huge worldwide cancer industry must

frequently release news about exciting new cancer remedies. Some of these widely promoted new treatments are in the early stages of being tested on human patients, some are not yet being tested on humans but are promoted as the new 'wonder' cure for cancer on the basis of animal experiments, and some are talked about as new 'breakthroughs' largely or even exclusively on the basis of a scientist's theory or hypothesis. These news stories may raise hopes falsely and they certainly distract attention and finance from those areas which really need it (the prevention of cancer and the care of those who have cancer) but they serve the purpose: they raise money for the cash hungry cancer industry.

When someone rattles a collecting tin in front of you and tells you that they need just a few more pence in order to continue with their breakthrough research it is difficult to say 'no'. It is hardly surprising that the worldwide cancer industry's income continues to grow at a quite phenomenal rate.

But has the cancer industry really made any noticeable progress in the fight against cancer? I don't think so. The mortality figures show that more people are dying from cancer now than were a generation ago. One in three people already have, or will develop, cancer. According to journal 'The Sciences' (published by the New York Academy of Sciences) the American Cancer Society has estimated that over half a million Americans were killed by cancer in 1994 alone - twice as many as were killed in the Second World War. Figures from around the world show that the picture is much the same everywhere.

In the nineteenth century cancer was a relatively uncommon disease. According to Garrison's History of Medicine it showed an 'alarming increase' in the early part of the twentieth century. Today, as I have already mentioned, cancer is firmly positioned (along with heart disease and stroke) as one of the three big 'killers' of the westernised world. The fact that the incidence of cancer has increased dramatically during the 20th century confirms, in my opinion, the view that cancer is, to a large extent, a man-made disease, created largely by our changing diet and our addiction to tobacco. In America it has been estimated that around $110 billion a year is spent on cancer. That is more than ten% of American's entire health care bill. But, in America, during the last fifteen years or so the incidence of cancer has steadily risen as has the number of people dying of cancer.

Writing in the European Medical Journal in Winter 1993 Dr Jack Tropp pointed out that 'despite the billions of dollars spent each year for cancer research and treatment, using the traditional methods of choice: surgery, chemotherapy and radiation therapy, in the overall picture nothing has changed in the mortality rates in the last thirty five years.'

Scientists who have assessed the value of the war against cancer agree that we are losing the fight and that there is no evidence to suggest that decades of expensive research have had much, if any, effect on the most fundamental measure of success or failure - death. In areas where cancer has become more amenable to treatment, or now takes fewer lives, it is usually changes in life style which are responsible - not discoveries made in the laboratory.

In my view, the effort, the money, and all the misguided hope and faith that has been poured into cancer research by the international cancer industry simply hasn't worked. And there are, I believe, several reasons why those fighting the war against cancer are losing. In my book 'Paper Doctors' (published in 1977) I complained that: 'Medical researchers involved in publicly or charitably financed cancer research persist in looking for the 'magical cure'. Much laboratory work has been started on the mistaken assumption that there is one disease called 'cancer' and that there will be a 'cure' which will enable doctors to treat all the patients suffering with cancer. Many projects have been funded because organisers (both qualified and lay) have believed that they might solve the problem of cancer once and for all to the well publicised credit of everyone concerned. Much money has undoubtedly been wasted on research which has duplicated work done elsewhere and which has moved in directions unlikely ever to prove of practical benefit.'

These complaints, made nearly twenty years ago, are now

frequently echoed by other observers. In the March/April 1995 issue of The Sciences Robert N Proctor, associate professor of the history of science at Pennsylvania State University and author of 'Cancer Wars: How Politics Shapes What We Know and Don't Know About Cancer', pointed out that: 'Part of the problem lies in the very belief that a single, universal cure can be found.'

CHAPTER TWO

WOMEN GOLFERS AND BREAST CANCER

Breast cancer is one of the most constantly publicised and most greatly feared forms of cancer. Because of its very nature it is a type of cancer which arouses much emotion. Newspapers, magazines and medical journals have for decades been full of articles describing new forms of treatment. The medical journal, 'The Lancet', in an editorial, commented that: 'If one were to believe all the media hype, the triumphalism of the profession in published research, and the almost weekly miracle breakthroughs trumpeted by the cancer charities, one might be surprised that women are dying at all from this cancer.'

But women are still dying from breast cancer. Indeed, the overall death rate from breast cancer hasn't changed and isn't changing. Despite all the talk and all the promises the number of women dying from breast cancer hasn't altered. The disease is still as great a killer as it was decades ago. 'In the two short decades since Nixon began shovelling money into the NCI (National Cancer Institute) breast cancer has claimed more U.S. lives than the Vietnam War, the Korean War, World War I and World War II put together,' reported the journal 'Australian Health and Healing'.

The real tragedy is that since 1942 there has been steadily accumulating evidence to show that there is link between breast cancer and dietary fat. I have little doubt that breast cancer could have been turned into a relatively uncommon disease, instead of one of the major killers of women, if politicians and doctors had been prepared to take on the food industry - and force the cancer industry to spread the truth. In Britain around 15,000 women die of breast cancer every year. That is roughly one every half hour. And yet if those women had known which foods to eat - and which to avoid - half of them could still be alive today.

Despite the quantity of epidemiological evidence which is available to show that fat and breast cancer are linked (and I have detailed some of this evidence later in this book) no one really knows the mechanism through which fat causes cancer. One theory is that synthetic chemicals - commonly used in the manufacture of pesticides - concentrate in the fatty tissues of animals. People who then eat animal after animal absorb the synthetic chemicals into their own bodies - and the concentrations of chemicals steady rise. (In an average lifetime a meat eater will consume 36 pigs, 36 sheep, 750 chickens and turkeys and several cows.)

According to the journal 'Australasian Health and Healing' more than 177 organochlorines (synthetic chemicals created when chlorine gas is bonded to carbon-rich organic matter) have been found in the tissues of the general population of the United States and Canada. Organochlorines can cause infertility, birth defects, miscarriages, immune system suppression, metabolic dysfunction, behavioural disorders, hormonal abnormalities and cancer.

These chlorine based compounds can cause cancer in various ways. Some cause cancer directly. Others produce cancers by interfering with or mimicking human hormones. A third group suppress the immune system and then enhance the carcinogenic effect of other chemicals. These chemicals seem to strike first at the reproductive system - which is probably why a heavy fat consumption increases the risk of developing cancers of the breast, prostate, and uterus.

We accept chemicals because they make our life easier and because the big chemical companies have become financially and politically powerful. We assume that they are essential and we assume that they are safe because that is what the big companies tell us. I don't believe they are essential and I certainly don't believe that they are safe.

Here is some of the epidemiological evidence collected by the journal 'Australasian Health and Healing' to support the theory that it is the chemicals in fat which cause cancer:

• Women in parts of America which were routinely blanketed with aerial sprayings of the pesticide DDT during the 1950s have one of the highest rates of breast cancer in America.

• Female chemical workers who were exposed to high levels of dioxin in a German pesticide plant had double the cancer mortality rate of the German population - and higher than average rates of breast cancer. A study in America produced similar results.

• Women professional golfers, most of whom played the game every day, have a high rate of breast cancer. It is suspected that these women may have been poisoned by the chemicals with which most golf courses are saturated.

• According to a U.S. Environmental Protection Agency study parts of America with hazardous waste sites were 6.5 times as likely to have a high breast cancer rate than other areas.

• Women who have breast cancer have high blood levels of the chemicals suspected of causing the disease. A study of 229 women from New York City showed that women who developed breast cancer had substantially higher levels of suspected pesticide chemicals.

• During the early 1970s breast cancer rates in Israel were among the highest and fastest rising in the world. In 1978 Israel phased out the use of several suspected pesticides. Doctors noted that the levels of chemicals in breast milk dropped quite quickly. The incidence of breast cancer duly started to fall too.

The medical profession, which has largely ignored the existing evidence showing what causes breast cancer, does, of course, remain willing to continue to make money out of 'treating' women who develop breast cancer.

Perhaps the biggest irony of all is the fact that although doctors have widely ignored the available evidence that would have enabled them to protect most of their patients from breast cancer (and, indeed, many other major types of cancer too) they have devised their own high technology form of preventive medicine - the mammogram. It is difficult not to be cynical about this. Teaching women how to reduce their chances of developing breast cancer (and teaching them how to check their own breasts for lumps) would earn the profession very little. So the medical profession - and its companion industries - have introduced a high technology form of cancer detection so that they can make money out of the general anxiety about breast cancer.

In my book 'The Health Scandal' (which was published in 1988 and violently attacked by doctors all over the country - even though some of those who were doing the attacking admitted that they hadn't actually bothered to read it) I objected to mammography screening partly on the grounds that there must surely be risks in having regular X ray examinations (it would, I thought, be hideously, grotesquely ironic if a new technique designed to spot breast cancer at an early stage turned out, twenty years later, to cause breast cancer) and partly on the grounds that a mass breast screening programme simply would not work and would not make a significant difference to the number of women dying of breast cancer because the interval of one year between examinations was too long.

I pointed out that the available evidence showed clearly that self examination was much better and more effective (as well as being considerably cheaper and requiring far less highly trained medical manpower).'A proper educational programme' I wrote, 'designed to teach British women how to examine their breasts properly, would undoubtedly have a dramatic effect on the number of women dying from breast cancer in Britain. It would cost very little and it would produce continuing results. But it would not, of course, provide work for the unwanted radiologists who we are training every year. And it probably wouldn't satisfy the strident spokeswomen who believe that annual screening clinics must be better than regular checks done at home.'

At the time this comment was dismissed as heresy by many in the medical establishment who argued that self screening was useless. But seven years later more doctors began to question the logic of mammography. And by then several studies had suggested that the radiation accumulated through yearly mammograms might actually be causing breast cancer.

The ultimate irony is, perhaps, the fact that the pharmaceutical industry makes billions of dollars selling drugs for the treatment of cancers which some believe may have been created by its billion dollar sister industry: the chemical industry.

Doctors and drug companies and the cancer industry have come together in the belief that the best way to deal with cancer (or to prevent it) is to use drugs.

Consider, for example, the tamoxifen experiment - designed to find out if the drug tamoxifen will protect women from breast cancer.

The drug tamoxifen isn't new. For several years it has been given to women who are suffering from breast cancer and as a treatment for infertility. The risks associated with this drug may be low enough to justify its use when a woman's life is threatened because she already has cancer but just look at the possible side effects which may be associated with tamoxifen: hot flushes, vaginal bleeding, gastro-intestinal pain, light headedness, skin rash, cataracts, fluid retention, and endometrial carcinoma.

I suppose the politicians and scientists who are in charge of this extraordinary trial may claim that women won't mind feeling light headed, developing a skin rash and cataracts and suffering from hot flushes and vaginal bleeding if there is a chance that they will be less likely to get breast cancer but what about that last hazard: endometrial carcinoma? Endometrial carcinoma is cancer of the uterus!

So, according to the existing information we already have about this drug we know that there is a possible risk that it may cause cancer of the uterus. And governments around the world have given researchers approval to give it to thousands of perfectly healthy women to see if it stops them getting breast cancer.

What sort of inspired lunacy is this?

These healthy women - some of them still in their 30s - will be given the drug to take for several years.

Those who are organising the trial are also ignoring evidence showing that tamoxifen causes liver cancer in rats and gonadal cancer in mice. As I have been arguing for years animal experiments cannot be used to predict what will happen when a drug is given to human patients. (Though some readers may, like me, find it a little difficult to understand why the cancer industry still supports animal experiments when it is insisting that the tamoxifen experiments on animals be ignored so that they can go ahead with this human experiment!).

If those who are organising the trial are satisfied with the results doctors around the world will, presumably, be encouraged to prescribe tamoxifen on a regular basis for millions of healthy women. The drug could quickly become one of the most profitable pharmacological products ever marketed.

CHAPTER THREE

ANIMAL EXPERIMENTS: MORE HARM

THAN GOOD

Raising money for cancer research is now big business. But it is a business that has, if you assess its effectiveness critically, surely been a dismal failure. One of the main reasons why the cancer industry has failed is because it has concentrated too much of its massive effort on animal experiments. Millions of pounds have, for example, been spent on giving cancer to animals - when it has for years been widely appreciated that the cancers animals get are quite different to the cancers people get.

Animal experiments are cheap, relatively easy to perform, do not require great skills, and tend to produce some sort of result very quickly. Cancer research workers often work together with big drug companies which love animal experiments for their double edged value. If an experiment on a group of animals shows that a drug does not harm those particular animals then the drug company will use the evidence to ensure that the drug is given a clean bill of health around the world. But if an experiment on a group of animals shows that the drug does harm those particular animals the drug company will ignore the evidence on the grounds that animals are different to people - and that the results are, therefore, of no significance. As the American Committee on Diet, Nutrition and Cancer (of the U.S. National Research Council) pointed out in 'Diet, Nutrition and Cancer' (published by the National Academy Press): 'animals are not human, and the etiology of the cancers they develop may not duplicate that for cancers in humans.' The United States Surgeon General has pointed out: 'an important weakness is that virtually all animal studies test single, genetically uniform (inbred) strains of one or two non-human species under highly uniform conditions of diet, temperature, stress, exposure to infectious diseases etc.'.

Cancer researchers frequently claim that if animal experiments are banned they will never be able to find a cure for cancer. The choice, they say to those who dare to question what they do, is simple: the lives of a few animals (since animal researchers around the world kill around one thousand animals every thirty seconds this is something of an under-statement) or the lives of your children. This crude and, sadly, often effective blackmail (which I have previously described as a form of intellectual terrorism) presupposes that cancer researchers are eventually going to find a 'cure' for cancer - indeed, it assumes, quite illogically, that there can ever be a single cure for the two hundred quite different diseases which make up the group of illnesses we know as 'cancer'. There is no evidence at all to suggest that this is an accurate presupposition.

On the contrary the evidence clearly shows that animal experiments are a complete waste of time, that animal experiments have never led to any useful breakthroughs and that they are never likely to lead to any useful breakthroughs.

Animals get cancer, it is true, but the cancers they get are quite different to the types of cancer which affect human beings. Moreover, animals respond quite differently to the various types of treatment which are available. The results animal researchers obtain are totally without value. The total uncertainty makes the whole business valueless.

Instead of helping doctors find a cure for cancer those scientists who do experiments on animals have held back medical progress and have been responsible for much pain and distress and hundreds of thousands of unnecessary deaths.

A standard test used on rats gives results which can be accurately applied to human beings just 38% of the time. This means that 62% of the time the results obtained through animal experiments are wrong. Since tossing a coin would give a long term 50% chance of accuracy it would clearly be quicker, more effective, more efficient and cheaper for these scientists to spend their working days sitting around tossing coins to assess the safety of chemicals. ('Heads! Yes! We can give this to patients! Tails! No! Patients can't take that one.')

But, in political and financial terms, tossing a coin would certainly not be as useful as using animals. Consider tobacco, for example. The link between tobacco and cancer was identified many years ago by doctors whose observations and research work had involved human patients. But long after doctors had established the link between tobacco and cancer in humans animal researchers were still forcing dogs to smoke and painting tobacco tar on the backs of mice in attempts to show whether or not there was a laboratory link between tobacco and cancer. Politicians who wanted to avoid taking action against the wealthy and big tax paying tobacco companies (the U.S. government receives more than $13 billion a year in tobacco tax) were able to do so on the grounds that they were still awaiting laboratory confirmation of the link between tobacco and cancer. Decades of vague, inconclusive and contradictory results have enabled the world's tobacco industry to keep selling a product which is responsible for approximately one third of all cancer deaths and which, over the years, must have been responsible for more deaths, disease and misery than any other product ever invented.

Using animals to test new anti-cancer drugs is equally absurd. 'Test beds' made of human tissue cells are available. These can be used reliably to test anti-cancer drugs. I cannot see the scientific sense in testing a drug on animals when it can be tested on cells which are identical to those within the patients who will take the drug. (There is, as I have already pointed out, coarse commercial sense in doing such tests on animals. If a test on one species show that a drug is lethal the test can be repeated on another species, and another, and another until a more promising or acceptable result is obtained. The drug can then be launched worldwide as suitable for human patients.)

Animal experiments are useless because animals are completely different to people. According to Dr Irwin Bross, giving evidence to the United States Congress: 'conflicting animal results have often delayed and hampered the war on cancer, they have never produced a single, substantial advance either in the prevention or treatment of human cancer.' The medical journal The Lancet commented that: 'since no animal tumour is closely related to a cancer in human beings an agent which is active in the laboratory may well prove useless clinically.'

CHAPTER FOUR

THE SEARCH FOR A MAGIC CURE

Huge amounts of money have been spent on searching for a 'magic' cure for cancer when cancer is scores of different diseases and it is as unlikely that we are going to find a single cure for all these different diseases as we are to find a single drug that will cure all infections.

Most of the effort expended by the cancer industry has been spent on searching for the ever elusive cure. And yet throughout medical history it has consistently been clear that it is invariably easier and more effective (and, in the long run a great deal cheaper) to prevent illness than it is to try and cure it. The evidence shows quite clearly that any improvements in life expectancy which have taken place in the last few decades have been almost entirely due not to the development of a sophisticated and expensive medical profession or the development of a highly profitable international pharmaceutical industry but to simple improvements in living standards. The enemies of death and disease in the last century have not been doctors and drug companies but (relatively) clean drinking water and better sewage facilities. There has been a modest improvement in life expectancy in the last hundred years but it has been nothing to do drugs or surgery.

(And the improvement has been more modest than most doctors like to admit. Figures for the UK are difficult to get hold of because the British Government traditionally regards every health care statistic as a state secret, to be shared only with the pharmaceutical industry, but figures published by the United States Bureau of Census show that 33% of people born in 1907 could expect to live to the age of 75. Later figures show that 33% of the people born in 1977 could expect to live to the age of 80. Remove the improvements produced by better living conditions, cleaner water supplies, and the reduction in deaths during or just after childbirth and it becomes clear that doctors, drug companies and hospitals cannot possibly have had any useful effect on life expectancy. Indeed, the figures show that there has been an increase in mortality rates among the middle aged and a dramatic increase in the incidence of disorders such as diabetes, arthritis, heart disease and cancer.)

The truth is that the inventor of the flush lavatory has saved a million times more lives than any doctor. It is not the men who discovered antibiotics - and who now prescribe them with such reckless over-enthusiasm - whom we should thank for the virtual disappearance of some of the best known killer infectious diseases of the nineteenth century but the men who dug our sewers and laid the first water pipes.

We don't need more money pouring into cancer research because we already know what causes most forms of cancer.

According to one honest observer: 'Basic cancer research is an excellent slush fund for molecular biologists but it won't have any impact on cancer'. Cancer is created by chemical pollutants, by unhealthy, fatty, food and by tobacco. Poisoned water supplies, dangerous prescription drugs and the over use of X rays have also contributed to the incidence of cancer.

With immune systems constantly battered by polluted air,

adulterated and chemically impregnated food and a constant onslaught from the drugs we buy for ourselves, or allow our doctors to prescribe for us, it is not surprising that increasing numbers of people succumb to one of the many different types of cancer. We know what causes 80% of all cases of cancer! Eight out of ten people who develop cancer could have been saved if money and effort had been put into prevention.

I do not believe that any wonder cure for cancer will come

from the 'cancer industry'. But if I had the annual income the cancer industry enjoys I believe that I could turn cancer into a historical oddity within five years.

'Why,' you may ask, 'do the big cancer charities not spend

more money on trying to prevent cancer? Why do they spend all their money gambling on the chance of finding a 'cure' when they could save millions of lives simply by using their resources to publicise what we already know?'

To understand the answers to these questions you have to understand that the cancer research industry is exactly that -an industry. It is a massive, worldwide multi-billion dollar industry which employs hundreds of thousands of scientists and administrators. Sadly, I suspect that for many of these employees the search for a cure for cancer has become the end instead of just the beginning. Much of the cancer industry is run by and for scientific researchers. The cancer industry needs a constant stream of dollars to keep its laboratories running. If the cancer industry spent its income on explaining to people how to avoid cancer there would be little or no place for research laboratories and a great many scientists would be put out of work. Worse still, if the cancer industry reduced the number of people dying of cancer its own income would fall.

Besides, those whose job it is to raise money know that it is far easier to persuade people to put money into a collecting tin if you tell them that they are contributing towards the search for a cure. Persuading people to avoid known causes of cancer is thankless work. Not many people will put money into your collecting tin if you stand on a street corner handing out leaflets containing sensible eating advice. It is much easier to raise money if you talk about new breakthroughs and show photographs of people (preferably children or pretty young women) who are dying of cancer. It is much easier to raise a million dollars for a new piece of machinery than it is to raise a million dollars for a print bill or a television advertising campaign designed to explain to people how they can avoid cancer.

Those who are responsible for raising money for the cancer industry know that it is impossible to personalise a preventive medicine campaign because you can never show a picture of a man, woman or child whose life has been saved by a leaflet. A picture of a child who is dying of cancer and who is waiting for laboratory scientists to find a cure will attract far more public support than an appeal for funds to help save unnamed individuals in the future.

And so it is the scientists searching for laboratory cures who get the big grants and the prestigious awards and who are fussed over and praised by the politicians and the journalists. Scientists who have established links which would enable us to save millions of lives by organising effective prevention programmes are as unlikely to win Nobel prizes as are doctors who devote themselves to teaching the principles of healthy living to millions of people.

My book 'Paper Doctors' (which was published in 1977) contained a whole chapter attacking the cancer industry in some detail. 'According to the International Agency for Research on Cancer 80% of all cancers are environmentally induced,' I wrote. 'The Director of the American National Cancer Institute has publicly stated that he believes that the figure is nearer to 90%. Eric Boyland, for many years Professor of Biochemistry at the Institute of Cancer Research in London, concluded after careful consideration of all the likely causes of cancer that between 85 and 90% of all human tumours are caused by chemicals. According to the 1974/5 Annual Report of the Medical Research Council, 'This statement has never been seriously challenged and the experience and research of the past few years has tended to support this view of cancer."

Further on in the same chapter in 'Paper Doctors' I went on to say: 'If we took the advice given by the experts who quote these figures then we could cut down cancer rates dramatically. However, the emphasis among cancer researchers is very much on finding a cure rather than reducing the incidence of cancer. Since there is so much evidence to show that the causative factors can be controlled (cigarettes cause one third of all cancers in the United Kingdom) that is, to say the least, surprising. There are many explanations for this disastrous misreading of the needs within cancer research. Undoubtedly one important reason is that there are thousands of surgeons, radiologists and radiotherapists (not to mention pure researchers) earning their living from traditional methods of cancer research and control. Public health is not a fashionable speciality within the medical profession: there is no opportunity for private practice and a general lack of financial incentive for brighter physicians to enter this field. And of equal importance perhaps is the fact that industrial pressures all oppose attempts to control the causative factors responsible for the vast majority of cancers.'

'I doubt if you will even think of all this next time you see a stranger shaking a collecting tin at you and asking for a contribution towards cancer research. You, like everyone else, have been lured over the years into believing the claims and promises made by honestly motivated but misdirected cancer researchers. If you really cared about cancer patients you would give your money to a caring organisation or an anti-smoking campaign, taking note of Lord Zuckerman's conclusion in his official Report entitled 'Cancer Research' published in 1972. He wrote that 'a sudden increase in funds for cancer research could not be effectively used' and suggested that money be spent on helping those struggling to look after relatives dying of cancer rather than on more research programmes.'

But those words fell on deaf ears.

In the two decades since 'Paper Doctors' was published the cancer industry has continued to grow. It is now a multi- billion dollar industry. The cancer researchers are still pleading for more money; they are still claiming that if they are given enough money they will be able to find a cure for cancer. It is, they suggest, only a shortage of money which is responsible for the continuing rise in the incidence of cancer deaths. They want to do more animal experiments and they have now devised animals which are genetically bred to develop cancer.

Cancer industry employees say they want to know 'how' cancer develops. They don't seem to understand that the 'how' is not important. It would be nice to know the 'how'. But it is the 'why' which matters. And we already know why cancer is a major killer.

By insisting on finding the 'how' the cancer industry is playing right into the hands of the industries which are responsible for much cancer. The tobacco industry is constantly calling for more research into the links between tobacco and cancer. By calling for more research the tobacco industry is able to make itself look good - and at the same time to suggest that there is still some controversy and doubt about the links between tobacco and cancer.

But we don't need more research into tobacco and cancer.

Everything we need to know can be summed up in four words: 'Smoking tobacco causes cancer.' And all we need now is a proper programme to persuade people not to smoke. But governments continue to spend millions fighting an unwinnable 'drugs war' against the users of cannabis, cocaine and heroin while they subsidise tobacco - a much more dangerous drug.

The tobacco industry isn't the only industry trying to cause

confusion and doubt. Every cancer producing industry follows the same route: campaigning for more and more research and claiming that it is research and not prevention which will lead to a solution.

Consumers look on, puzzled and bemused. They quite like the idea of someone finding a 'cure' for cancer because it means that they can carry on eating the fatty food they like and smoking the cigarettes they enjoy without having to worry. 'Had such campaigns been at work during the Irish potato famine, which killed more than a million Irish between 1845 and 1849, they might have engendered studies into the biomechanical processes of famine rather than the social forces that gave rise to it,' wrote Robert N Proctor in 'The Sciences'. 'Investigators would have secured funding for research into why starvation ran in families instead of examining the social Darwinist policies of the Manchester era or studying how Ireland was able to export grain even during the worst years of the famine.'

In 1977 I estimated that the annual total expenditure on cancer research was probably close to $1,500,000,000. Today the annual total expenditure on cancer research is so vast as to be immeasurable. The National Cancer Institute, in the United States of America, has, by itself, spent more than $29,000,000,000 on cancer research! There are very few industries in the world which have grown as rapidly as the cancer industry - the charities, laboratories, research scientists and administrators who are devoted to the search for a cure for cancer.

The real irony in all this is the fact that money really isn't the key issue. You cannot 'buy' successful research. Although the huge cancer industry has spent a vast portion of its massive budget on laboratory research designed to find a 'magic' cure for cancer the majority of the most important and dramatic discoveries about cancer have been made by observant practitioners who have devoted themselves to a study of human patients and their habits.

In 'Paper Doctors' I explained: 'One of the most important breakthroughs in cancer research of recent years was made, not by researchers in expensive institutes but by a practising British surgeon, Denis Burkitt, working in Uganda. His first research grant from Government funds totalled £15 and his second, for £150, came from the Medical Research Council and was spent on an old jeep. By logical, patient study Burkitt managed to map the occurrence of a tumour common in children in that part of the world. He matched the map he prepared with other factors and eventually managed to show that the cancer was probably caused by a virus. Eventually he learnt how to cure the tumour. So, one of the most important discoveries was made, not by a professional researcher but by an observant doctor happy to continue his studies in his own time and at his own expense. Too many doctors these days are unwilling to begin any research programme unless they are first financed by an official agency and properly recognised as bona fide research workers.'

CHAPTER FIVE

LOSING THE WAR

The cancer industry uses statistics to try to persuade us that their billions of dollars are well spent. But I believe that if you look at the statistics objectively and carefully they show exactly the opposite. Although there have been a few (very well publicised) steps forward against some individual (and usually relatively rare) forms of cancer the overall effect has been negligible. I have absolutely no doubt at all that if the money spent on cancer research had been spent on teaching people what we know about the causes of cancer millions of lives would have been saved.

We are not winning the war against cancer. The war against

cancer has been as badly fought as the war against 'drugs'. And it has been as unsuccessful.

Despite all the publicity and the extravagant claims the war

against cancer (officially declared by President Richard Nixon in 1971) has been a failure. In the spring of 1995 the journal 'The Sciences' reported that: 'Five year survival rates for the majority of cancers (lung, colon, breast and stomach cancers, for example) remain essentially what they were twenty years ago.'

Leading American physicians and scientists have described the war against cancer as 'a medical Vietnam' and (rather more colourfully) 'a bunch of shit'.

In 1977, when my book 'Paper Doctors' was published I reported that 130,000 people died each year from cancer in England and Wales. By 1986 the figure had reached 140,000. The latest official figures from the government show that the figure has been going up steadily since then - it is now around 142,000.

'There have been some very important advances in cancer

treatment over the last three decades,' said Dr John Bailar, who had been a researcher at the National Cancer Institute in the United States of America and had been, for years, editor in chief of the 'Journal of the National Cancer Institute'. 'But with respect to the cure of cancer, they are limited largely to the cancers that tend to occur in children and young adults, and those make up only perhaps one or two% of the total cancer burden.'

'Cancer deaths rates continue to go up year after year,' said Dr Bailar. 'Now these are real increases. I've taken out the effect of the changing size of the population, the changing age structure, declining mortality from other disease and we look at what's left. There is a genuine increase in the frequency of deaths from cancer, and this has been going on quite steadily for a number of years now.'

'Unfortunately, we have only managed to impact on the more rare malignancies with little effect on the most common and deadly forms of cancer,' says Dr Neal D Barnard in his book 'The Power of Your Plate'.

The cancer industry deliberately draws attention to its modest successes in the treatment of a few, relatively uncommon childhood cancers (putting pictures of children whose lives have been saved onto promotional leaflets and posters is a sure-fire way to keep the money pouring in). But there has been little or no improvement in the death rates for the big killers: lung cancer, breast cancer, prostate cancer or cancer of the colon.

None of this explains why governments have also failed to

teach their citizens the facts about cancer. However, there are, I believe, two explanations for this seemingly mysterious state of affairs. Firstly, governments are always wary of annoying big, powerful, tax paying industries and there is absolutely no doubt that many huge, international corporations would be (to put it mildly) exceedingly upset if millions of potential consumers were warned of the dangers of smoking, eating or drinking exceedingly profitable substances. Secondly, (and I offer no excuse for the fact that this sounds extremely cynical) governments do not want people to live longer. On the contrary, they have a vested interest in people not living too long. People who live on into their sixties, seventies and eighties have to be given pensions and cost governments a great deal of money.

If you find all this difficult to accept just remember that although it is widely and generally accepted that thirty% of cancers are caused by tobacco, governments and Europe still subsidise the growing of tobacco. Governments spend a tiny amount of money trying to persuade their citizens not to smoke. But they spend one hundred times as much on helping the tobacco companies keep down the price of their products. It would, in health terms, be more sensible for these governments to subsidise the growing and distribution of cocaine. It is as crazy as if governments were giving muggers and bank robbers grants for the purchase of masks, guns and getaway cars.

'Americans worry a great deal about the import of cocaine

and other drugs into the U.S.,' pointed out one observer sadly, 'but little is done to halt the far more deadly export of tobacco into the nations of the third world.'

Nor is it easy to explain why the medical profession has failed to teach people how to avoid cancer. Doctors have known the causes of most cancers for nearly two decades but very few indeed have done anything to help spread the word among their patients.

But the vast majority of doctors have for years earned their

living by attending to the sick. Most doctors hardly, if ever, think about doing anything to keep their patients healthy. For example, I doubt if many people reading this book have ever discussed diet or nutrition with their doctors - even though a good, balanced diet is an essential prerequisite for good health.

Some doctors may make a little effort to persuade those patients who have already fallen ill to give up or cut down smoking but such efforts are usually superficial and rather uninspired. The trouble with doctors is, as I explained at some length in my book 'Betrayal of Trust', that the medical profession has been bought by the pharmaceutical industry. Doctors are educated by the drug industry and controlled by it and the result is that when the average doctor thinks of 'patients' he or she thinks of 'drugs'. Doctors are taught to respond to illness by prescribing a bottle or packet of pills. Many doctors (probably a large majority of doctors) have been so brainwashed by the powerful international drugs industry that they regard any treatment which does not involve drugs as hocus pocus and they regard preventive medicine as trivial, irrelevant nonsense which is rather beneath them.

Instead of putting energy and effort into teaching their patients which foods to avoid (and which to eat) doctors have created 'medical screening' - a high tech solution to a low tech problem.

The principle is a simple one: the patient goes to the doctor and the doctor (for a fee, of course) does tests which are designed to spot early signs of cancer or other diseases. But screening doesn't work. A once a year check up is no alternative to a healthier lifestyle.

In several earlier books I have explained why I neither approve of nor support the principle of medical screening programmes. But, as an example, consider, the much publicised cervical screening programme.

Cervical screening programmes have, over the years, consistently attracted huge budgets. Untold thousands of doctors have been employed in performing cervical smears. The laudable aim of the cervical screening programme is to reduce the number of women dying from cervical cancer and it is constantly being argued that if more money were put into cervical screening programmes thousands of lives could be saved.

But I don't think the evidence supports this contention. Indeed, a cynical observer might be inclined to suggest that it is the medical profession which has benefited most from the cervical screening programme. One leading physician described the results of screening for cervical cancer in Britain as 'disappointing'. A writer in one leading international journal argued that: 'there is no clear evidence that this screening is beneficial, and it may well be doing more harm than good'.

Although doctors around the world have strongly argued in

favour of more expenditure on smears they have never really decided exactly which women need to be targeted. There have never been any trials done which show the undisputed value of the cervical screening programme. Amazingly, I don't believe that the cervical screening programme has ever been properly evaluated.

Indeed, the evidence rather firmly shows that cervical screening programmes may not be of any real value to women as a whole - though there will, of course, always be individual women who are able to say that their lives were 'saved'.

Statistically, the test has not been shown to save lives in any country where it has been introduced. Huge amounts of public money have been spent on organised cervical screening programmes but the incidence of cervical cancer has hardly altered in thirty years. In countries where the incidence of cervical cancer and the number of women dying from it are falling the rate of decline is no greater than it was before the screening programme began.

Britain, for example, has been spending over £150 million a year on its cervical screening programme since the early 1980s but the programme has never been based on any logical plan and the number of women dying from cervical cancer has hardly changed. In fact because the disease is relatively uncommon (cervical cancer does not make the top ten list of causes of death among women) huge numbers of women who do not have the disease have been subjected to unnecessary tests. Because 'false positives' are fairly common, many women are referred unnecessarily for further tests and treatment. Even more worryingly 'false negatives' occur in between 7% and 60% of smears. An enormous amount of anxiety and fear are produced by the often inefficient and thoughtless way in which smear reports are submitted to patients.

The failure of the screening test to save lives is not difficult to understand when you look at the available evidence.

The first major problem is that smear tests are neither accurate nor reliable. Different cytologists reading the same slide may produce entirely different results. Abnormal cells may be present in one sample and not in another from the same woman.

Most worrying is the fact that many of the smears taken by

doctors are useless. One report concluded that ten% of all cervical smears sent to cytology departments were useless and a further forty% were of limited usefulness in detecting carcinoma of the cervix. The main problems are that doctors either take smears from the wrong site or use faulty techniques.

There is, surprisingly perhaps, still a considerable amount of confusion about the natural history of the cervical cancer.

From the evidence that is available it seems that some slow growing cancers do regress if left alone while fast growing cancers develop so rapidly that smears would have to be done every few months to be of real value.

One report concluded that a third of the biopsies performed

because of positive cervical cytology are likely to have been performed for lesions which are insignificant or would have disappeared if left alone. Since biopsies are performed under anaesthetic (with which there is always a risk of death) it seems perfectly possible that the dangers associated with having a smear just may be greater than the possible advantages.

There is, not surprisingly, an enormous amount of confusion among gynaecologists about the best way to treat cervical cancer even when it has been identified.

Sadly, even when useful smears are taken the laboratories providing results cannot always cope. And even when laboratories do discover significant changes women are not always notified. Delays mean that at least one woman has died after having had a positive smear but before she had been given her results.

The enthusiasm of doctors for cervical screening is not difficult to understand. Sadly, I suspect that the medical profession's enthusiasm for cervical screening may, in part at least, have been aroused by and sustained by the profit to be made, rather than any belief that cervical screening will save lives. In one medical journal a writer pointed out that: 'unless doctors take urgent individual action a serious breakdown in cervical smear recalls - affecting GP income - could arise in five years' time.'

If the money spent on doctors' fees had been spent on educational programmes it is just possible that many thousands more lives could have been saved. For example, how many women know that it is vitally important that they should see their doctors straight away if they notice any intermenstrual bleeding, any bleeding after sex or any abnormal or unusual vaginal discharge.

LIFESTYLE ASSESSMENT

Do this simple quiz to assess your cancer risk:

1. Do you smoke:

a) a lot

b) average

c) a little

d) not at all

2. How many hours a week do you spend in close proximity to people who smoke:

a) over 80 hours

b) 40 to 80 hours

c) 10 to 40 hours

d) less than 10 hours

3. Do you make an effort to cut your intake of fatty food?

a) yes - a big effort

b) yes - a small effort

c) no - not at all

4. Do you eat butter?

a) yes

b) no

5. Do you drink full fat milk?

a) yes

b) no

6. Do you eat full fat cheese?

a) yes

b) no

7. Do you eat red meat?

a) yes

b) no

8. Do you eat white meat?

a) yes

b) no

9. Do you eat fish?

a) yes

b) no

10. How much alcohol do you drink?

a) more than one or two drinks a day

b) one or two drinks a day

c) less than one or two drinks a day

11. How much processed, packaged food do you eat:

a) a lot

b) hardly any/none at all

12. Do you eat smoked or barbecued food?

a) yes

b) no

13. Do you eat salt cured or salt pickled food?

a) yes

b) no

14. How much fibre do you think your diet contains?

a) a lot

b) an average amount

c) not much

15. Does your diet regularly include citrus fruits (oranges, grapefruit, lemons)?

a) yes

b) no

16. Do you regularly eat other fruits (e.g. apples, bananas, pineapple, apricots, peaches, strawberries)?

a) yes

b) no

17. Does your diet regularly include cabbage, broccoli, cauliflower or Brussels sprouts?

a) yes

b) no

18. Do you regularly eat garlic and/or onions?

a) yes

b) no

19. Does your diet regularly include whole grains (unrefined rice, wheat, oats, barley)?

a) yes

b) no

20. Does your diet regularly include potatoes?

a) yes

b) no

21. Does your diet regularly include carrots?

a) yes

b) no

22. Does your diet regularly include these vegetables: spinach, peas, beans, lentils, tomatoes, asparagus, kale?

a) yes

b) no

23. Are you noticeably overweight?

a) yes

b) no

24. Do you take care to restrict your exposure to sunlight?

a) yes

b) no

25. Do you take care to avoid spending too much time close to electricity wires and electrical appliances?

a) yes

b) no

26. Do you live near to a nuclear power station?

a) yes

b) no

27. Does your job put you in contact with any chemicals which might cause cancer?

a) yes

b) no

28. On average, do you have more than one X ray a year?

a) yes

b) no

29. Do you regularly take prescribed medicines?

a) yes

b) no

30. Do you regularly take medicines you buy yourself?

a) yes

b) no

Now check your score:

1. a) 50 b) 30 c) 10 d) 0

2. a) 3 b) 2 c) 1 d) 0

3. a) 0 b) 10 c) 20

4. a) 2 b) 0

5. a) 2 b) 0

6. a) 2 b) 0

7. a) 10 b) 0

8. a) 2 b) 0

9. a) 2 b) 0

10. a) 5 b) 0 c) 0

11. a) 1 b) 0

12. a) 1 b) 0

13. a) 1 b) 0

14. a) 0 b) 5 c) 10

15. a) 0 b) 3

16. a) 0 b) 2

17. a) 0 b) 10

18. a) 0 b) 3

19. a) 0 b) 10

20. a) 0 b) 1

21. a) 0 b) 1

22. a) 0 b) 3

23. a) 7 b) 0

24. a) 0 b) 3

25. a) 0 b) 1

26. a) 1 b) 0

27. a) 4 b) 0

28. a) 5 b) 0

29. a) 3 b) 0

30. a) 1 b) 0

The higher your score the greater your risk of developing cancer.

If you scored 100 or more then your cancer risk is danger- ously high

If you scored between 75 and 99 your cancer risk is very high - well above average.

If you scored between 50 and 74 your cancer risk is average.

If you scored between 25 and 49 your cancer risk is below average.

If you scored between 1 and 24 your cancer risk is con- siderably lower than average.

If you scored 0 your cancer risk is very low

If you want to reduce your cancer risk - read the

remainder of this book!

CHAPTER ONE

FOODS THAT CAUSE CANCER -

AN A to Z of EVIDENCE

You may be surprised to hear that there is now clear medi- cal and scientific evidence available to show that nothing, not even tobacco, influences your chances of developing cancer as much as the food you choose to eat. It is estimated that between 30% and 60% of all cancers are caused by what you choose to eat. Doctors, scientists and supporters of the cancer industry claim that the battle against cancer will only be won with the aid of more money. They claim that in order to obtain the information we need we must spend, spend, spend. But that isn't true. It is not more knowledge we need (we have, as I pointed out in 'Paper Doctors' nearly twenty years ago, already amassed far more knowledge than we will ever use in our lifetime), but the ability and courage and determination to use the knowledge we already have.

Back in 1982 the National Research Council in the United

States of America published a technical report entitled Diet, Nutrition and Cancer which showed that diet was probably the single most important factor in the development of cancer, and that there was evidence linking cancers of the breast, colon and prostate to particular foods or types of food.

It is a scandal of astonishing proportions that a majority of the population still do not know about these vitally important and well established links. It is an even bigger scandal that a majority of the medical profession are unaware of these links too. Most doctors I have spoken to - even recently qualified ones - still dismiss the idea of a food/cancer link as mumbo-jumbo nonsense, preferring to rely entirely on prescription drugs, radiotherapy and surgery as 'treatments' for cancer. The average medical student probably spends more time staring down a microscope at histology slides than he or she spends studying the importance and significance of nutrition.

After he had seen a previous book of mine (Food For Thought) advertised as containing material describing the links between food and cancer one doctor sent me a very indignant letter, asking me whether I thought it was ethical to publish such statements as 'Between a third and a half of all cancers are caused by eating the wrong types of food' and 'You can dramatically reduce your chances of developing cancer of the breast, cancer of the prostate, cancer of the colon, cancer of the ovary or cancer of the uterus.'

(To be fair, after I had sent him details of a small amount of the evidence upon which I had based those statements, the doctor wrote back and apologised.)

I checked one large (over 1,000 pages) recently published medical textbook and found that the chapter on cancer summed up the role of food as a causal agent in just one, rather short sentence. I find this all extremely difficult to understand. I have been studying scientific research papers for over two decades and I have never seen such convincing research as that which shows the links between particular types of food and particular types of cancer.

It is not uncommon for new drugs to be launched after clinical trials which may have involved relatively small numbers of patients. In my book Betrayal of Trust I pointed out that the number of patients studied in clinical trials before a drug is marketed is, on average, only 1,480 - and that the final, total, overall figure is sometimes much less than this.

In contrast some of the individual research projects which have been published showing links between food and cancer have involved tens of thousands of patients!

I have absolutely no doubt that if these undeniable links had been publicised by the responsible authorities (in medicine as elsewhere the phrase 'responsible authorities' is, I fear, oxymoronic) countless millions of lives and an enormous amount of agony and distress could - and would - have been avoided.

The suppression of this information by a greedy and conscience-free food industry, compliant revenue conscious politicians, a cancer industry dominated by grant hungry researchers and an uncaring, drug company dominated medical profession has, I sincerely believe, led to more deaths than any war in history.

Since the early 1980s the amount of evidence linking diet to cancer has grown steadily. In 1990 even the British Medical Association, hardly an organisation which would be widely described as revolutionary, supported the view that there is a link between food and cancer. Their published view was that 35%, just over a third, were caused by the natural constituents of food and that another 1% of cancers were caused by food additives.

Other organisations suggest that the link between food and

cancer is even higher. The National Academy of Sciences in the United States, founded in 1863 by Act of Congress to serve as an official adviser to the US government in all matters of science and technology, has reported that researchers have estimated that almost 60% of women's cancers and a little more than 40% of men's cancers are related to nutritional factors.

Because I recognise that many readers may be sceptical about the claim that there is a strong link between diet and food (such scepticism will undoubtedly be enhanced by the fact that neither governments nor the medical profession have made much, if any, effort to publicise these links) I have in this chapter summarised just some of the scientific evidence which supports this claim.

My previous efforts to publicise the health values of eating

a low fat, meatless diet have been consistently confronted by scepticism and opposition. It is my hope that this summary of just some of the most important available evidence supporting the contention that certain foods are a risk factor in the development of cancer will help settle this particular controversy permanently and may, perhaps, help other writers who may be tempted to tell their readers the truth about food and cancer.

I must emphasise that this list is not intended to be comprehensive. I have accumulated a list of several hundred scientific papers and journal articles dealing with the links between food and cancer (and many thousands more dealing with food and other disorders) and this list is merely intended to be representative - and to provide you with some idea of the breadth and importance of the available evidence.

If you would like to study even more evidence linking food

to cancer (and many other diseases) then I heartily recommend the book 'Nutritional Influences on Illness: A sourcebook of clinical research', written by Melvyn R. Werbach MD and published by Thorsons. In a remarkably comprehensive and quite fascinating book Dr Werbach has listed and summarised thousands of clinical studies and scientific reports. He reports that: 'The cancers most closely associated with nutritional factors are breast and endometrial cancer in women, prostate cancer in men and gastrointestinal cancer', points out that 'the value of a low fat, high fibre diet in cancer prevention is well documented' and notes that 'avoidance of smoked, pickled and salt cured foods' has been shown...to be beneficial in preventing cancers of the gastrointestinal tract.'

Dr Werbach adds that 'there is early evidence that certain pre-cancerous changes may be reversible with supplementation'. He reports that cervical dysplasia may possibly be reversed with folic acid supplements, that calcium supplements may help 'reduce the number of rapidly proliferating cells in the colonic epithelium in patients with family histories of colon cancer and elevated numbers of such cells compared to controls' and that 'vitamin A or beta-carotene reduce the percent of genetically damaged cells inside the cheek when betel quid, a tobacco like plant mixture is chewed regularly.'

Exhibit A:

Title: Calorie-Providing Nutrients and Risk of Breast Cancer

Authors: Paolo Toniolo (Epidemiology Unit, Istituto Nazionale per lo Studio e la Cura dei Tumori,

Milan, Italy and Department of Environmental Medicine, New York University Medical Center, New York, USA), Elio Riboli (Unit of Analytical Epidemiology, International Agency for Research on Cancer, Lyon, France), Fulvia Protta (Pathology Service, Ospedale Maggiore S. Giovanni, Turin, Italy), Martine Charrel (Unit of Analytical Epidem- iology,International Agency for Research on Cancer, Lyon,France), Alberto M Cappa (Pathology Service, Ospedale Maggiore S.Giovanni, Turin, Italy)

Source: Journal of the National Cancer Institute

Date: February 15th 1989

Report: This study, which was conducted in the province of Vercelli in northwestern Italy where there is a moderately high incidence of breast cancer, was designed to investigate the role of diet in breast cancer and 'to test the primary hypothesis that fat and proteins from animal sources are associated with increased risk of breast cancer.'

The researchers questioned 250 women with breast cancer and a random sample of 499 women from the general population, using a structured questionnaire to identify the types and quantities of food each woman consumed.

After reporting that they had 'found evidence that the intake of total fat, saturated fat or proteins of animal origin is positively associated with the risk of breast cancer in women' the researchers reported that their 'findings had suggested that during adult life a reduction in total fat to less than 30% of calorie intake, of saturated fat to less than 10% of calorie intake and of animal proteins to less than six% may lead to a substantial reduction in the incidence of breast cancer in population subgroups with high intake of saturated fat and animal proteins in agreement with some dietary recommendations that have been made.'

'These data,' said the researchers, 'suggest that during adult life a reduction in dietary intake of fat and proteins of animal origin may contribute to a substantial reduction in the incidence of breast cancer in population subgroups with high intake of animal products.'

Exhibit B

Title: Dietary Habits and Prognostic Factors in Breast Cancer

Authors: Lars-Erik Holm (Department of Cancer Preven- tion, Karolinska Hospital, Stockholm, Sweden), Eva Callmer (Department of Medical Nutrition, Huddinge Hospital, Huddinge, Sweden), Marie- Louise Hjalmar (Department of General Oncology, Danderyd Hospital, Danderyd, Sweden), Elizabet Lidbrink (Department of General Oncology, South Hospital, Stockholm, Sweden), Bo Nilsson (Depart ment of Cancer Epidemiology, Karolinska Hos-

pital, Stockholm, Sweden, Lambert Skoog (Dep- artment of Clinical Cytology, Karolinska Hospital, Stockholm, Sweden).

Source: Journal of the National Cancer Institute

Date: August 16th 1989

Report: The link between dietary fat and breast tumours was first described in 1942. Since then many other scientists have published papers detailing this link in further detail. 'Dietary fat has been suggested as an etiological factor in human breast cancer because of the high correlation between national per capita fat consumption and breast cancer incidence,' begin the authors of this paper whose study included 240 women who had surgery for breast cancer between the years of 1983 and 1986 in Stockholm, Sweden. All the women involved in this study were aged between 50 and 65 at the time that their cancers were diagnosed. After analysing their results the authors of this paper concluded: 'The results of this study of mainly postmenopausal women and some premenopausal women suggest that dietary habits have an impact on the prognosis of breast cancer.'

'Study results indicate that dietary factors may act in breast carcinogenesis,' continued the authors in their discussion of their results. 'Several studies have found that these factors also operate after diagnosis of the tumour, suggesting that dietary habits are important even after a successful primary treatment. The results of this study suggest that dietary patterns of the western world (eg high fat intake and low consumption of carbohydrates and fiber) affect certain prognostic factors in breast cancer, such as tumour size and ER (oestrogen receptor) content of the tumour.'

Exhibit C

Title: A Case-Control Study of Prostatic Cancer With Reference to Dietary Habits

Authors: Kenji Oishi, Kenichiro Okada, Osamu Yoshida, Hirohiko Yamabe, Yoshiyuki Ohno, Richard B Hayes and Fritz H. Schroeder; (these authors were from the Department of Urology, Kyoto University, Kyoto, Japan; Laboratory of Anatomic Pathology, Kyoto University Hospital, Japan; Department of Public Health, Medical School, Nagoya City Uni- versity, Nagoya, Japan; Department of Urology,

Faculty of Medicine, Erasmus University Rotterdam, The Netherlands.)

Source: The Prostate

Date: 1988

Report: In 1950 the incidence of prostatic cancer in Japan was about 0.4 per 100,000 male members of the population but by 1963 it had increased to 2.0 per 100,000 and by 1975 it was 2.5 per 100,000.

Observers have suggested that this increase (which is not unique, for increases in the incidence of cancer of the lung, cancer of the breast and cancer of the colon have also occurred in recent years) may be linked to the westernization of Japanese eating habits. During recent years the consumption of fat, animal protein, eggs, dairy products and oil have all increased considerably in Japan.

This study was of sufferers from prostatic cancer from 1981 to 1984 and patients suffering from benign prostatic hypertrophy (non cancerous prostate enlargement) and was conducted to find the risk factors for prostatic cancer. 'Two well trained nutritionists and one urologist interviewed each subject at the time of hospital admission'.

The findings included: 'Low daily intake of beta-carotene ... were significantly correlated with prostatic cancer development.'

Exhibit D

Title: Relation of Meat, Fat and Fiber Intake To The Risk of Colon Cancer in a Prospective Study Among Women

Authors: Walter C. Willett M.D, Meir J.Stampfer M.D., Graham A. Colditz M.D., Bernard A. Rosner Ph.D., and Frank E. Speizer M.D. from the Channing

Laboratory, Department of Medicine, Harvard Medical School and Brigham and Women's Hos-

pital, the Department of Preventive Medicine, Harvard Medical School and the Departments of Epidemiology, Nutrition and Biostatistics at the Harvard School of Public Health, all in Boston in the United States of America.

Source: The New England Journal of Medicine

Date: December 13th 1990

Report: The authors of this paper began by pointing out that in Western countries cancer rates are up to ten times as high as they are in many Far Eastern and developing countries. For many years doctors and scientists have noted rapid increases in rates of colon cancer among men and women migrating from low risk areas to high risk areas and these observations have suggested that the large differences which exist may be due to environmental causes rather than genetic causes.

The authors pointed out that two general dietary hypotheses have evolved in recent decades: firstly, that dietary fat, particularly from animal sources, increases the risk of colon cancer and secondly, that the intake of fiber reduces the risk.

This study involved observing 88,751 women between the ages of 34 and 59 for six years. The women, none of whom had any history of cancer, inflammatory bowel disease or familial polyposis, completed a questionnaire about their eating habits in 1980. By the year 1986 a total of 150 cases of colon cancer had been noted.

The researchers concluded that 'animal fat was positively associated with the risk of colon cancer' but that 'no association was found for vegetable fat'. The researchers found that women who ate beef, pork or lamb as a main dish every day were more likely to develop cancer of the colon than were women who ate beef, pork or lamb as a main dish less than once a month. It was also found that processed meats and liver with also 'significantly associated with increased risk'.

'The ratio of the intake of red meat to the intake of chicken and fish was particularly strongly associated with an increased incidence of colon cancer,' concluded the researchers, who also noted that 'a low intake of fiber from fruits appeared to contribute to the risk of colon cancer, but this relation was not statistically independent of meat intake.'

'These prospective data provide evidence for the hypothesis that a high intake of animal fat increases the risk of colon cancer,' said the researchers, 'and they support existing recommendations to substitute fish and chicken for meats high in fat.'

Exhibit E

Title: Dietary Fat Consumption and Survival Among Women With Breast Cancer

Authors: David I. Gregorio, Department of Social and Pre- ventive Medicine, State University of New York, Buffalo, New York, United States of America; Law rence J.Emrich, Department of Biomathematics, Roswell Park Memorial Institute, Buffalo, New York, United States of America; Saxon Graham, Depart- ment of Social and Preventive Medicine, State

University of New York, Buffalo, New York, United States of America; James R.Marshall, Department of Social and Preventive Medicine, State University of New York, Buffalo, New York, United States of America, and Takuma Nemoto, Department of Breast Surgery, Roswell Park Memorial Institute, Buffalo, New York, United States of America

Source: JNCI

Date: July 1985

Report: The researchers estimated monthly fat consumption for a total of 854 patients who completed dietary intake interviews when they were admitted to hospital and subsequently estimated that 24% of the women consumed between 500 and 1,000 grams of fat a month, 42% consumed between 1,001 and 1,500 grams of fat a month, 21% consumed between 1,501 and 2,000 grams of fat a month and 11% consumed between 2,001 and 3,000 grams of fat.

They reported their findings as follows: 'Consistent with our hypothesis, an effect of fat intake on survival time was reported in this study' and concluded that the 'estimated risk of death at any time increased 1.4 fold for every 1,000 gram in monthly fat intake'.

Exhibit F

Title: Dietary Factors and Breast Cancer Risk

Authors: Jay H. Lubin, Environmental Epidemiology Branch, National Cancer Institute, Bethesda, Maryland, U.S.A.; Patricia E. Burns, Cross Cancer Institute, Edmonton, Alberta, Canada; William J. Blot,

Environmental Epidemiology Branch, National Cancer Institute, Bethesda, Maryland, U.S.A.; Regina G. Ziegler, Environmental Epidemiology Branch, National Cancer Institute, Bethesda, Mary- land, U.S.A.; Alan W.Lees, Cross Cancer Institute, Edmonton, Alberta, Canada and Joseph F. Fraumeni, Jr., Environmental Epidemiology Branch, National Cancer Institute, Bethesda, Maryland, U.S.A.

Source: International Journal of Cancer

Date: 1981

Report: These researchers questioned 577 women aged between 30 and 80 (all of whom had breast cancer) and 826 disease free women about their eating habits. They found that women who ate beef, pork and sweet desserts were significantly more likely to develop breast cancer than women who did not. They also found that women who fried with butter or margarine, as opposed to vegetable oils, and who used butter at the table were also more likely to develop breast cancer.

Exhibit G

Title: Role of Life-style and Dietary Habits in Risk of

Cancer among Seventh-Day Adventists

Author: Roland L. Phillips, Department of Biostatistics and Epidemiology, Loma Linda University School of Health, Loma Linda, California, U.S.A.

Source: Cancer Research

Date: November 1975

Report: Seventh-Day Adventists neither drink alcohol nor smoke, most avoid the use of coffee, tea, hot condiments and spices and about half eat a vegetarian diet which includes dairy produce and eggs. In his summary the author noted that existing data on cancer mortality in Seventh-Day Adventists showed death rates that were 50% to 70% of the rates within the general population for most of the cancer sites that are unrelated to smoking or drinking alcohol. The author's studies showed statistically significant links between the eating of beef, lamb or a combined group of highly saturated fat foods and the development of colon cancer. 'It is quite clear,' he wrote, 'that these results are supportive of the hypothesis that beef, meat and saturated fat or fat in general are etiologically related to colon cancer.' He went on to say that: 'Green leafy vegetables that are quite high in fiber are negatively associated.'

The author concluded:'Overall, the currently available evidence on cancer among Seventh-Day Adventists is consistent with the hypothesis that one or more components of the typical Adventist life style account for a large portion of their apparent reduced risk of the types of cancer which are unrelated to cigarette smoking and alcohol consumption. Aside from abstinence from smoking and drinking, the most distinctive feature of the typical Adventist life-style is a unique diet whose principal feature is lacto-ovo-vegetarianism.' The author then added that other researchers had shown that the typical lacto-ovo-vegetarian diet contained about 25% less fat and 50% more fibre than the average non-vegetarian diet.

Exhibit H

Title: Diet as an Etiological Factor in the Development of Cancers of the Colon and Rectum

Author: Margaret A. Howell, National Cancer Institute,

National Institutes of Health, Bethesda, Maryland, U.S.A.

Source: Journal of Chronic Diseases

Date: 1975

Report: The author concludes: 'The evidence suggests that meat, particularly beef, is a food associated with the development of malignancies of the large bowel.'

Exhibit I

Title: Nutrient Intakes In Relation to Cancer Incidence in Hawaii

Authors: L. N. Kolonel, J. H. Hankin, J. Lee, S.Y. Chu, A.M.Y. Nomura and M. Ward Hinds - from the Epidemi- ology Program, Cancer Center of Hawaii, Univer- sity of Hawaii, Honolulu, Hawaii, U.S.A.

Source: British Journal of Cancer

Date: 1981

Report: For this study 4,657 adults from the five main ethnic groups in Hawaii (Caucasians, Japanese, Chinese, Hawaiians and Filipinos) were interviewed about their diets between the years 1977 and 1979. The researchers reported that: 'significant positive associations were found for six of the cancer sites: breast cancer with fat (saturated, unsaturated, animal and total) and protein (animal); corpus-uteri cancer with the same components as breast cancer; prostate cancer with fat (saturated, animal) and protein (animal, total), stomach cancer with fat (fish only) and protein (fish only), lung cancer with cholesterol and laryngeal cancer with cholesterol. The researchers also found significant negative associations between breast and corpus-uteri cancers and carbohydrate intake.

Exhibit J

Title: Environmental Factors of Cancer of the Colon and Rectum

Authors: Ernest L. Wynder M.D., and Takao Shigematsu M.D., both from the Division of Environmental Cancerigenesis, Sloan Kettering Institute for

Cancer Research, New York, U.S.A.

Source: Cancer

Date: September 1967

Report: The authors of this paper concluded: '...dietary factors appear to be associated with the etiology of cancer of the large bowel. The dietary pattern that may fit the distribution of cancer of the large bowel includes a high intake of fats. The effect of this pattern appears to be more marked for cancer of the colon than cancer of the rectum.' And they added: 'There is a statistically significant association of obesity to cancer of the large bowel from the cecum to the sigmoid colon in men.'

Exhibit K

Title: Epidemiological Correlations between Diet and ' Cancer Frequency

Author: Pelayo Correa, Louisiana State University Medical Center, New Orleans, Louisiana, U.S.A.

Source: Cancer Research

Date: September 1981

Report: The author stated that: 'Strong and consistent correlations are reported between death rates of cancers of the colon and breast and the per capita consumption of total fat and of nutrients derived from animal sources, especially beef, pork, eggs and milk. Similar but less consistent correlations have been reported with cancers of the prostate, ovary and endometrium.' The author also reported that: 'Negative correlations of colon cancer rates and vegetable consumption are reported...Epidemiological data are consistent with the hypothesis that excessive beef and low vegetable consumption are causally related to colon cancer.' The author explained the link between the foods linked to cancer and the development of cancer by stating that 'these food items probably do not have a direct carcinogenic role but rather provide a microenvironment favourable to the actions of carcinogens.'

Exhibit L

Title: Food Consumption and Cancer of the Colon and Rectum in North-Eastern Italy

Authors: Ettore Bidoli, Epidemiology Unit, Aviano Cancer Center, Via Pedemontana Occ, Aviano, Italy; Silvia Franceschi, Epidemiology Unit, Aviano Cancer Center, Via Pedemontana Occ, Aviano, Italy and European Cancer Prevention Organisation, Epi-

demiology and Cancer Working Group, Brussels, Belgium; Renata Talamini, Epidemiology Unit, Aviano Cancer Center, Via Pedemontana Occ, Aviano, Italy; Salvatore Barra, Epidemiology Unit, Aviano Cancer Center, Via Pedemontana Occ, Aviano, Italy; and Carlo La Vecchia, Mario Negri

Institute for Pharmacological Research, Via Eiritrea, Milan, Italy and Institute of Social and Preventive

Medicine, University of Lausanne, Lausanne,

Switzerland

Source: International Journal of Cancer

Date: 1992

Report: The authors studied 123 patients with colon cancer, 125 patients with rectal cancer and 699 patients with no cancer. They concluded: '...the present study gives support for a protective effect associated with a fiber rich or vegetable rich diet, while it indicates that frequent consumption of refined starchy foods, eggs and fat rich foods such as cheese and red meat is a risk factor for colo-rectal cancer.' The authors found that a high consumption of margarine 'exerted a significant protection against cancer of the colon' and that 'high consumption of carrots, spinach, whole grain bread and pasta' reduced the risk of rectal cancer.

Exhibit M

Title: Diet and Lung Cancer in California Seventh-Day Adventists

Authors: Gary E.Fraser, Center for Health Research, Lorna Linda University, Lorna Linda, California, U.S.A., W.Lawrence Beeson, Center for Health Research, Lorna Linda University, Lorna Linda, California, U.S.A. and Roland L.Phillips, Lorna Linda Univer- sity, Lorna Linda, California, U.S.A.

Source: American Journal of Epidemiology

Date: 1991

Report: The authors reported that: 'fruit consumption was the dietary constituent that showed a strong, statistically significant protective association with lung cancer...'

Exhibit N

Title: Dietary Habits and Past Medical History as Related to Fatal Pancreas Cancer Risk Among Adventists Authors: Paul K. Mills Ph.D., W.Lawrence Beeson, M.S.P.H., David E.Abbey Ph.D., Gary E. Fraser M.D.,Ph.D., and Roland L. Phillips M.D., DrPH, from the Dep- artment of Preventive Medicine, School of Med-

icine, Lorna Linda University, Lorna Linda, Cali-

fornia, U.S.A.

Source: Cancer

Date: 1988

Report: The authors began by pointing out that the foods and/or nutrients which had been suggested to be associated with an increased risk of cancer of the prostate included 'total fat intake, eggs, animal protein, sugar, meat, coffee and butter' whereas the consumption of 'raw fruits and vegetables' had been 'consistently associated with decreased risk'. In this study the authors found that there was strong evidence that 'increasing consumption of vegetarian protein products, beans, lentils and peas as well as dried fruit' helped to protect against cancer of the pancreas.

Exhibit O

Title: Increasing use of soyfoods and their potential role in cancer prevention

Authors: Mark Messina Ph.D, Diet and Cancer Branch,

Division of Cancer Prevention and Control,

National Cancer Institute, Bethesda, U.S.A.

Virginia, Messina PhD, RD., Washington, U.S.A.

Source: Journal American Diet Association

Date: July 1991

Report: Most of the soybeans produced are used as animal feed but evidence has been accumulating that soybeans can prevent cancer. The authors of this report state that: 'Soybeans contain, in relatively high concentrations, several compounds with demonstrated anticarcinogenic activity.'

Exhibit P

Title: Dietary Prevention of Breast Cancer

Authors: David P. Rose and Jeanne M. Connolly, Division of Nutrition and Endocrinology, American Health Foundation, Valhalla, New York, U.S.A.

Source: Med.Oncol & Tumour Pharmacother.

Date: 1990

Report: 'A review of the epidemiological and experimental data suggests that dietary modification does have a place in breast cancer prevention,' say the authors. 'Based on present evidence, a dietary approach to breast cancer prevention should include weight control, when indicated, a reduction in dietary fat intake to approximately 20% of total fats, and an increase in fiber consumption to 25-30 grams a day.'

Exhibit Q

Title: Shift From a Mixed Diet to a Lactovegetarian Diet: Influence on Some Cancer-Associated Intestinal Bacte rial Enzyme Activities

Authors: Gunnar K. Johansson, Ludmila Ottova and Jan-

Ake Gustafsson (the authors are affiliated with the Department of Medical Nutrution, Karolinska In stitute, Huddinge University Hospital, Huddinge, Sweden).

Source: Nutr. Cancer

Date: 1990

Report: The authors of this paper conclude: '...the results in this paper indicate that a change from a mixed diet to a lactovegetarian diet leads to a decrease in certain enzyme activities proposed to be risk factors for colon cancer.'

Exhibit R

Title: Nutritional Approach to Oesophageal Cancer in Scotland

Author: Valda M Craddock, M.R.C. Toxicology Unit, Carshalton, Surrey, U.K.

Source: The Lancet

Date: 24th January 1987

Report: This publication took the form of a letter. The author began by pointing out that oesophageal cancer has a sharply defined geographical distribution in Britain - with many sufferers being in the north-west of Scotland where the consumption of green vegetables is believed to be low. The author also pointed out that oesophageal cancer is also very common in areas of China, in Iran, around the Caspian Sea and in parts of South Africa and adds that in all these regions the diet is low in fresh fruit and green vegetables - and the micronutrients they contain. 'A consistent finding from studies around the world of diet in relation to cancer is that consumption of fresh green vegetables is negatively associated with cancer,' writes the author. 'Deaths in women from this exceptionally distressing malignant disease total around 2,000 per year. Early diagnosis is not yet possible and treatment is unsatisfactory. Prevention, however, may be relatively easy.' The author also adds that the incidence of oesophageal cancer in Britain has almost doubled since 1970 and that 'there are epidemiological and experimental grounds for intervention studies which might lead to effective preventive measures, but nothing is being done.'

Exhibit S

Title: Cohort Study of Diet, Lifestyle and Prostate

Cancer in Adventist Men

Authors: Paul K. Mills, Ph.D, MPH; W. Lawrence Beeson, MSPH; Roland L.Phillips, M.D. DrPH, and Gary E.Fraser M.D., Ph.D; from the Department of Pre- ventive Medicine, Lorna Linda University School of Medicine, Lorna Linda, California, U.S.A.

Source: Cancer

Date: 1989

Report: For this study the authors evaluated dietary and lifestyle characteristics of approximately 14,000 Seventh-Day Adventist men. The men completed a detailed lifestyle questionnaire in 1976 and were monitored for cancer incidence until the end of1982. The authors concluded that: 'increasing consumption of beans, lentils and peas, tomatoes, raisin, dates and other dried fruit were all associated with significantly decreased prostate cancer risk.'

Exhibit T

Title: A Prospective Study of Dietary Fat and Risk of

Prostate Cancer

Authors: Edward Giovannucci, Channing Laboratory, De- partment of Medicine, Harvard Medical School and Brigham and Women's Hospital, Boston,

Mass, U.S.A.; Eric B. Rimm,Department of Epide- miology, Harvard School of Public Health, Boston, U.S.A.; Graham A. Colditz, Channing Laboratory, Department of Medicine, Harvard Medical School and Brigham and Women's Hospital, Department of Epidemiology, Harvard School of Public Health, Boston, U.S.A.; Meir J. Stampfer, Channing Labo- ratory, Department of Medicine, Harvard Medical School and Brigham and Women's Hospital, De- partment of Epidemiology, Harvard School of Public Health, Boston, U.S.A.; Alberto Ascherio, Depart ment of Nutrition, Harvard School of Public Health, Boston, U.S.A.; Chris C. Chute, Department of Health Sciences Research, Mayo Medical School, Rochester, Minn., U.S.A.; Walter C. Willett, Channing Laboratory, Department of Medicine, Harvard Medical School and Brigham and Wom- en's Hospital, Department of Epidemiology and

Department of Nutrition, Harvard School of Pub- lic Health, Boston, U.S.A.

Source: Journal of the National Cancer Institute

Date: October 6th 1993

Report: The authors pointed out that: 'The strong correlation between national consumption of fat and national rate of mortality from prostate cancer has raised the hypothesis that dietary fat increases the risk of this malignancy.' By studying information relating to 51,529 American men between the ages of 40 and 75 and sending follow up questionnaires to the men in 1988 and 1990 they examined the relationship of fat consumption to the incidence of advanced prostate cancer and to the total incidence of prostate cancer.

The authors found that 'total fat consumption was directly related to risk of advanced prostate cancer' and that 'this association was due primarily to animal fat ... but not vegetable fat. Red meat represented the food group with the strongest positive association with advanced cancer.'

The authors concluded that :'The results support the hypothesis that animal fat, especially fat from red meat, is associated with an elevated risk of advanced prostate cancer.' They also noted that: 'These findings support recommendations to lower intake of meat to reduce the risk of prostate cancer.'

Exhibit U

Title: Risk of death from cancer and ischaemic heart

disease in meat and non-meat eaters.

Authors: Margaret Thorogood, senior research fellow, De- partment of Public Health and Policy, London School of Hygiene and Tropical Medicine, London, UK; Jim Mann, professor, Department of Human Nutrition, University of Otago, Dunedin, New

Zealand; Paul Appleby, research officer, Depart ment of Public Health and Primary Care, Univer sity of Oxford, Oxford, U.K.; Klim McPherson, professor, Department of Public Health and Policy, London School of Hygiene and Tropical Medicine, London, U.K.

Source: British Medical Journal

Date: 25th June 1994

Report: The aim of this research was to investigate the health consequences of a vegetarian diet by examining the twelve year mortality of vegetarians and meat eaters. The researchers reported: 'These data confirm the findings of previous studies that have shown a reduction in all cause, cancer and cardiovascular mortality among people who do not eat meat.'

The researchers showed a 'roughly 40% reduction in mortality from cancer in vegetarians and fish eaters compared with meat eaters' and also added that 'the fact that total mortality was about 20% lower in the non-meat eating group than the meat eaters is perhaps of greatest clinical importance.'

Exhibit V

Title: Risk Factors for Renal-Cell Cancer in Shanghai, China

Authors: Joseph K. McLaughlin, National Cancer Institute, Division of Cancer Etiology, Epidemiology and Biostatistics Program, Bethesda, MD, U.S.A; Yu- Tang Gao, Shanghai Cancer Institute, Department of Epidemiology, Shanghai, People's Republic of China; Ru-Nie Gao, Shanghai Cancer Institute, De partment of Epidemiology, Shanghai, People's Re public of China; Wei Zheng, National Cancer Insti tute, Division of Cancer Etiology, Epidemiology and Biostatistics Program, Bethesda, MD, U.S.A; Bu- Tian Ji, Shanghai Cancer Institute, Department of Epidemiology, Shanghai, People's Republic of China; William J.Blot, National Cancer Institute, Division of Cancer Etiology, Epidemiology and Biostatistics Program, Bethesda, MD, U.S.A; and Joseph F. Fraumeni Jr, National Cancer Institute, Division of Cancer Etiology, Epidemiology and Biostatistics Program, Bethesda, MD, U.S.A;

Source: International Journal of Cancer

Date: 1992

Report: The authors studied 154 patients with renal cell cancer and 157 controls. They reported: 'Elevated risks were observed for cigarette smoking...and for increasing categories of body weight and meat consumption, while reduced risks were seen for increasing categories of fruit and vegetable intake. An increase was also observed for regular use of phenacetin containing analgesics.' The authors also noted that these findings were 'consistent with earlier studies in Western countries and indicate that many of the same etiologic factors for renal cell cancer operate in low and high risk societies.'

Exhibit W

Title: Shift from a mixed to a lactovegetarian diet: influ ence on acid lipids in fecal water - a potential risk factor for colon cancer

Authors: Ulrika Geltner Allinger, Gunnar K.Johansson, Jan- Ake Gustafsson and Joseph J.Rafter; from the De partment of Medical Nutrition, Karolinska Institute, Huddinge University Hospital, Huddinge, Sweden Source: The American Journal of Clinical Nutrition

Date: 1989

Report: The authors concluded that: 'the consumption of a lactovegetarian diet may reduce certain risk factors of potential significance in colon carcinogenesis.'

Exhibit X

Title: NCI dietary guidelines: rationale

Authors: Ritva R.Butrum Ph.D., Carolyn K.Clifford Ph.D., and Elaine Lanza Ph.D from the Division of Cancer Prevention and Control, National Cancer Institute, National Institutes of Health, Bethesda, MD, U.S.A.

Source: The American Journal of Clinical Nutrition

Date: 1988

Report: The authors report that in 1986 it was estimated that 930,000 Americans would develop cancer and that 472,000 individuals would die of their cancer. The National Cancer Institute (the NCI), which aims to reduce cancer incidence, morbidity and mortality, 'believes that the potential for dietary changes to reduce the risk of cancer is considerable and that the existing scientific data provide evidence that is sufficiently consistent to warrant prudent interim dietary guidelines that will promote good health and reduce the risk of some types of cancer.'

The NCI suggests reducing fat intake, increasing fibre intake, including a variety of fruits and vegetables in the daily diet, avoiding obesity, consuming alcoholic beverages in moderation if at all and minimising the consumption of salt cured, salt pickled and smoked foods. The NCI believes that if these guidelines were followed there would be a 50% reduction in cancer of the colon and rectum, a 25% reduction in breast cancer and a 15% reduction in cancers of the prostate, endometrium and gallbladder.

Exhibit Y

Title: Shifting from a Conventional Diet to an Uncooked Vegan Diet Reversibly Alters Fecal Hydrolytic

Activities in Humans

Authors: Wen Hua Ling and Osmo Hanninen from the De- partment of Physiology, University of Kuopio, Kuopio, Finland

Source: Journal of Nutrition

Date: 1992

Report: The authors conclude that an 'uncooked extreme vegan diet causes a decrease in bacterial enzymes and certain toxic products that have been implicated in colon cancer risk.'

Exhibit Z

Title: Association between dietary changes and mortal ity rates: Israel 1949 to 1977; a trend-free regres- sion model

Author: Aviva Palgi Ph.D., Instructor in Nutrition, Harvard Medical School, Nutrition/Metabolism Laboratory, New England Deaconess Hospital, United States of America

Source: The American Journal of Clinical Nutrition

Date: August 1981

Report: The author investigated the statistical effect of 'changes in food consumption of the Israeli population during 1949 to 1977 on concurrent mortality rates from cancer, heart disease, peptic ulcer, and diabetes mellitus'.

The author reported that: 'All the investigated mortality rates were in statistically significant positive association with increasing total fat consumption. Mortality rates of ischemic heart disease as well as of hypertensive and cerebrovascular disease were in positive association with both plant fat and animal fat. These findings suggest that reduced total fat intake may prove to reduce the investigated mortality rates.'

Note 1: Where I have included medical or scientific qualifications alongside the names of researchers it is because these were published on the scientific papers concerned. Where no qualifications are listed it is because no qualifications were listed on the journal articles I have quoted.

Note 2: In January 1986 the 'Journal of Occupational Medicine' published a paper entitled 'Cancer Mortality Among White Males in the Meat Industry'. The paper was written by Eric S.Johnson M.B., B.S.; H. R. Fischman D.V.M.; Genevieve M. Martanoski M.D.; and E. Diamond Ph.D. from the Department of Epidemiology, The Johns Hopkins University School of Hygiene and Public Health, Baltimore, United States of America. The authors studied 13,844 members of a meat cutter's union from July 1949 to December 1980 'to examine cancer occurrence in the meat industry'. They reported that a 'statistically significant proportional mortality ratio of 2.9 was obtained for Hodgkin's disease among abattoir workers' and that 'the results suggest that the excess risk of death from Hodgkin's disease in abattoir workers may be associated with the slaughtering activity'. They also found that meat packing plant workers were more likely to develop bone cancer, cancer of the buccal cavity and pharynx and lung cancer than workers in other industries.

I report this paper because although it does not show a direct link between cancer and the eating of meat as a food it does pose an important question: if human beings can get cancer merely by handling meat why on earth should there be any surprise that human beings can get cancer from eating meat? The authors of this paper also named viruses which naturally cause cancer in cattle and chickens and pointed out that these viruses are present not only in diseased but also in healthy cattle and chickens destined for human consumption. 'Evidence suggests that consumers of meat and unpasteurized milk may be exposed to these viruses. It would appear, therefore, that these viruses present a potentially serious public health problem.' Other researchers have made similar discoveries about a link between the meat industry and the development of cancer. A study of 300,000 adult white males in Washington State in the United States of America showed a 'statistically significant elevated risk of death from cancer of the buccal cavity and pharynx among butchers and meatcutters'.

CHAPTER TWO

FOODS TO AVOID -

AND FOODS TO EAT

However careful you are to avoid potentially cancerous chemicals cancer cells will occasionally develop inside your body. Most of the time those cancer cells are dealt with speedily and effectively by your body's defence systems. White blood cells find and destroy cancer cells in just the same way that they find and destroy bacteria.

Your body's natural immune system (and its ability to deal with cancer) will be damaged if you eat the wrong sort of foods - and will be aided and improved if you eat the right foods. Fatty foods will weaken your immune system and make your body less capable of fighting off those occasional cancer cells. When researchers studied the blood of human volunteers they found that a low fat diet greatly improved the activity of the body's natural killer cells.

Incidentally, it has been shown that as far as the body's immune system is concerned vegetable fats are just as bad as animal fats. You will protect your heart by reducing your animal fat consumption but in order to protect yourself against cancer you need to reduce your entire fat consumption - and that includes vegetable oils.

Vegetarians have more than double the cancer cell destroying capability of non-vegetarians. But this is not entirely due to the low fat content of a vegetarian diet. It is probably also due to the fact that vegetarians consume fewer toxic chemicals and no animal proteins.

And vegetarians have another advantage too: the ability of the human body's natural, killer cells to do their work is improved by substances such as beta-carotene which are found in considerable quantities in vegetables. (One survey of meat eaters showed that many could neither name nor describe a any green vegetables.)

Foods to avoid

Fatty foods in general

The average diet still contains 40% fat. Many official recommendations are still only encouraging a reduction in fat intake to around 30% of the diet - despite evidence showing that a reduction to somewhere between 10% and 20% would make far more sense. Fat was the first dietary constituent to be linked with cancer and there is now probably more evidence damning fat than any other foodstuff. According to the National Academy of Sciences report 'Diet, Nutrition and Cancer' (published by the National Academy Press in Washington D.C., U.S.A. in 1982) there were at least six international studies published in the 1970s which showed a direct association between the amount of fat eaten and breast cancer incidence or mortality. In addition a state by state study published within the U.S. showed a significant direct correlation between fat intake and breast cancer mortality rates.

'Japanese women have the lowest breast cancer rate in the world,' says Dr Oliver Alabaster, Director of the Institute for Disease Prevention at the George Washington University Medical Center, quoted in 'The Power of your Plate', by Dr Neal D Barnard. 'Many Japanese women have migrated to Hawaii and the U.S. mainland. While marrying within their own community and keeping the population relatively unchanged genetically, they shifted their diet towards a more Western, higher fat diet and their breast cancer rate steadily climbed. A few decades ago the Japanese diet contained only around 10% fat - today the average Japanese diet contains about 25% fat. Within one generation it approximated that of Caucasian women living around them. This is very dramatic evidence that cancer is mainly environmentally induced, rather than genetically inherited.'

Peter Greenwald, Director of Cancer Prevention and Control of the National Cancer Institute in the United States of America also says that there is a great deal of evidence that fat increases cancer risk and points out that the differences in breast cancer rates in different countries cannot be due to other factors such as stress, pollution or industrialisation since there are highly industrialised countries such as Japan where stress and pollution are as high as the U.S.A. or Europe but where colon and breast cancer rates are low.

Since the mid 1970s there has been strong evidence to show

a link between a high fat intake and prostate cancer. The National Academy of Sciences reports that an American study showed a correlation between a high fat intake and a high risk for prostate cancer. Studies in 41 countries have shown a high correlation between mortality from prostate cancer and intake of fats, milk and meats (especially beef). A ten year Japanese study involving 122,261 men aged 40 or older showed 'an inverse association between daily intake of green and yellow vegetables and mortality from prostate cancer'. Another study showed that vegetarian men were less likely to develop prostate cancer.

Other studies also confirm this link. In 1993 a study of 47,855 men, reported in the 'Medical Research Modernization Committee Report' revealed that those men who had high fat diets had a relative risk of 1.79 for advanced prostate cancer compared to those on a low fat diet. The investigators found that 'most animal fats were associated with advanced prostate cancer, but fats from vegetables, dairy products (except butter) and fish were not.'

The National Academy of Sciences reports that: 'other reproductive organs for which there have been associations between dietary fat and cancer include the testes, corpus uteri, and ovary'.

There is also a considerable amount of published evidence available to show that there is a firm association between dietary fat and gastrointestinal tract cancer. Some researchers have shown a link between dietary fat and cancer of the pancreas, and others have shown a link between fat and stomach cancer. Researchers have also accumulated strong evidence confirming a correlation between dietary fat and large bowel cancer (cancers of the colon and rectum).

The National Academy of Sciences reports that: 'In general, it is not possible to identify specific components of fat as being clearly responsible for the observed effects, although total fat and saturated fat have been associated most frequently.'

With all this evidence available directly from observations of human patients it is exceedingly difficult to see why such a large proportion of the cancer industry's budget is spent on performing experiments on animals.

Although the evidence showing that fat causes cancer is totally convincing (a United States Surgeon General has advised U.S. citizens that 'a comparison of populations indicates that death rates for cancers of the breast, colon and prostate are directly proportional to estimated dietary fat intakes') there is still a considerable amount of doubt about the mechanism whereby fat causes cancer.

One theory is that carcinogenic chemicals simply dissolve and accumulate in fatty tissues. If this is the case then people who eat animal fats will suffer twice for the chances are high that the fat they are eating already contains dissolved carcinogens. Another possibility is that fat may encourage the development of cancer by affecting the activity of sex hormones. Vegetarian and low fat diets reduce the levels of circulating female sex hormones such as oestradiol. Sex hormones are known to help promote the development of breast cancer and cancer of the reproductive organs (such as uterus and ovary in women and the prostate in men).

Despite the lack of clear evidence about exactly how fat causes cancer the final message is quite clear - to reduce your cancer risk you should make a real effort to cut back your fat intake - and that includes cutting out vegetable fats too. You should not make the mistake of assuming that you can avoid or cut down your fat intake noticeably by living on a diet of chicken and fish. Although it is widely believed that both fish and chicken are low in fat the truth is that even skinless white meat from a chicken is 23% fat while most fish contain between 20 to 30% fat and some are much higher - mackerel, for example, contains over 50% fat. The only truly low fat diet is a diet which is mainly composed of vegetables, fruits, and whole grain cereals. Rice contains only about 1% fat and no plant foods contain any cholesterol (although frying potatoes and turning them into chips can add a lot of fat!)

If you ignore this message then you are making a clear and

conscious choice to accept a high cancer risk as the price for your high fat diet.

Meat and animal products

Numerous researchers have linked protein with cancers of the breast, prostate, endometrium (lining of the uterus), colon and rectum, pancreas and kidney. And the type of protein which is most likely to cause cancer is protein obtained from meat.

The United States Surgeon General's Report 'Nutrition and Health' said: 'In one international correlational study, for example, a positive association was observed between total protein and animal protein and breast, colon, prostate, renal and endometrial cancers (Armstrong and Doll 1975). Similarly, a migrant study indicated an association between meat consumption and cancer of the breast and colon (Kolonel 1987).'

The Surgeon General also reported that: 'Studies have also found an association between breast cancer and meat intake (Lubin et al 1981) and an association of meat, especially beef, with large bowel cancer among Japanese (Haenszel et al 1973)...'

One possible reason for the meat-cancer link may be the fact that chemicals such as DDT tend to accumulate in animal tissues - and may be found in animal tissues years after their usage has been controlled or stopped. Whether it is the chemicals in animal protein which cause cancer is, however, a question of rather theoretical interest: the important point is that meat causes cancer.

There is evidence to show that Japanese women who eat meat daily have more than eight times the risk of breast cancer compared to poorer women who rarely consume meat.

There have also been several reports showing a high correlation between meat (an important source of dietary fat, especially saturated fat) and colon cancer. Beef has been specifically named as one type of meat associated with colon cancer. Several studies have shown a relationship between the incidence of prostate cancer and the consumption of animal protein.

Because most people who eat a lot of meat usually also eat a great deal of fat (because meat often contains a lot of fat) it is difficult to know whether these links between meat and cancer are a result of the protein in the meat or the fat in the meat. It is also possible that the link between meat and cancer is a result of mutagens being formed during the cooking of meat. And some experts have pointed out that carcinogenic fat soluble contaminants such as drugs and pesticides may be the reason why meat causes cancer.

However, I regard the question of how meat causes cancer as being of largely theoretical interest. The important thing is that most of us need to eat less protein in general - and since there is a link between meat and cancer it seems pretty clear that cutting out meat is a sensible way to cut down protein.

Dairy products

The consumption of milk and other dairy products is relatively new in human history. And it is only in the 'highly developed' and 'westernised' world that milk drinking is considered essential or even normal.

Milk drinking (and the consumption of other dairy products) has developed because farmers and marketing experts have created the products and not because consuming these products is normal, essential or healthy. Babies need their mother's milk, taken direct from the breast, but no human beings need to drink dairy milk, or eat butter or cheese or other dairy products.

It is, almost certainly, the fat in dairy products which makes them particularly dangerous. Inevitably, therefore, some dairy products are more dangerous than others. Butter and cream and high fat cheeses are far more likely to cause problems than are low fat yoghurt, low fat cheese or skimmed milk.

But it isn't only the fat in dairy products which causes problems. There has for some years been accumulating evidence to suggest that dairy products can cause a wide variety of illnesses and there is now also some evidence to suggest that dairy products may be a contributory factor in the development of cancer of the ovary. The problem is, it seems, that a sugar in milk called lactose is broken down within the body to produce another sugar called galactose - which is then, in turn, broken down again. But if the consumption of dairy produce exceeds the body's ability to break down galactose this sugar may accumulate in the blood - and have an effect on the ovaries. Drinking low or non fat milk won't help this particular problem because the problem is caused by a sugar not fat.

Alcohol

It has been known for some years that there is an association between alcohol abuse and cancer. In 1937, in France, it was noted that 95% of patients with oesophageal cancer were alcohol abusers. Another French study, published in the 1960s and involving 4,000 patients, showed a significant correlation between alcohol consumption and cancers of the tongue, hypopharynx and larynx. In 1964 the World Health Organisation concluded that: 'excessive consumption of alcoholi